PMID- 15249795
OWN - NLM
STAT- MEDLINE
DA  - 20040713
DCOM- 20041216
PUBM- Print
IS  - 1524-6175 (Print)
VI  - 6
IP  - 7
DP  - 2004 Jul
TI  - Interview with Louis Tobian, MD. Interview by Marvin Moser.
PG  - 391-2
FAU - Tobian, Louis
AU  - Tobian L
LA  - eng
PT  - Historical Article
PT  - Interview
PL  - United States
TA  - J Clin Hypertens (Greenwich)
JT  - Journal of clinical hypertension (Greenwich, Conn.)
JID - 100888554
RN  - 0 (Diuretics)
RN  - 7440-23-5 (Sodium)
RN  - 7732-18-5 (Water)
SB  - IM
MH  - Biomedical Research/*history
MH  - Blood Vessels/chemistry
MH  - Diuretics/therapeutic use
MH  - History, 20th Century
MH  - Humans
MH  - Hypertension/drug therapy/*history/physiopathology
MH  - Sodium/analysis
MH  - Water/analysis
EDAT- 2004/07/14 05:00
MHDA- 2004/12/17 09:00
PST - ppublish
SO  - J Clin Hypertens (Greenwich) 2004 Jul;6(7):391-2.

PMID- 9931073
OWN - NLM
STAT- MEDLINE
DA  - 19990219
DCOM- 19990219
LR  - 20041117
PUBM- Print
IS  - 0194-911X (Print)
VI  - 33
IP  - 1
DP  - 1999 Jan
TI  - Story of the birth of the journal called Hypertension.
PG  - 7
FAU - Tobian, L
AU  - Tobian L
LA  - eng
PT  - Historical Article
PT  - Journal Article
PL  - UNITED STATES
TA  - Hypertension
JT  - Hypertension.
JID - 7906255
SB  - IM
SB  - Q
MH  - American Heart Association/*history
MH  - History, 20th Century
MH  - *Hypertension
MH  - Periodicals/*history
MH  - United States
EDAT- 1999/02/04
MHDA- 1999/02/04 00:01
PST - ppublish
SO  - Hypertension 1999 Jan;33(1):7.

PMID- 9160776
OWN - NLM
STAT- MEDLINE
DA  - 19970717
DCOM- 19970717
LR  - 20041117
PUBM- Print
IS  - 0895-7061 (Print)
VI  - 10
IP  - 5 Pt 2
DP  - 1997 May
TI  - Introduction to the Dahl symposium.
PG  - 27S-28S
AD  - Department of Internal Medicine, School of Medicine, University of
      Minnesota, Minneapolis 55455-0374, USA.
FAU - Tobian, L Jr
AU  - Tobian L Jr
FAU - Bintliff SJ
AU  - Bintliff SJ
LA  - eng
PT  - Journal Article
PL  - UNITED STATES
TA  - Am J Hypertens
JT  - American journal of hypertension :  journal of the American Society of
      Hypertension.
JID - 8803676
RN  - 7647-14-5 (Sodium Chloride)
SB  - IM
MH  - Animals
MH  - Diet
MH  - Humans
MH  - Hypertension/*chemically induced/*genetics
MH  - Rats
MH  - *Sodium Chloride/administration & dosage
EDAT- 1997/05/01
MHDA- 2001/03/28 10:01
PST - ppublish
SO  - Am J Hypertens 1997 May;10(5 Pt 2):27S-28S.

PMID- 9022555
OWN - NLM
STAT- MEDLINE
DA  - 19970226
DCOM- 19970226
LR  - 20051116
PUBM- Print
IS  - 0002-9165 (Print)
VI  - 65
IP  - 2 Suppl
DP  - 1997 Feb
TI  - Dietary sodium chloride and potassium have effects on the pathophysiology
      of hypertension in humans and animals.
PG  - 606S-611S
AB  - A diet high in NaCl can raise blood pressure in susceptible people and
      animals, probably by similar mechanisms. The possibly harmful effects of a
      high-NaCl diet are not unexpected because both prehistoric humans and
      mammals evolved in a low-NaCl environment. Evolutionary forces molded
      mammals to adapt well to a low sodium intake; modern high NaCl intakes go
      against this adaptation. A high-NaCl diet can cause premature mortality by
      raising blood pressure in susceptible people. We have new evidence that in
      hypertension, a high-NaCl diet can cause a great increase in mortality
      even though it does not cause a further blood pressure rise, partially
      because of multiple small cerebral infarcts. Recent evidence also
      indicates that a high-potassium diet reduces the rise of blood pressure
      caused by a high-NaCl diet, whereas a low-normal potassium intake
      encourages an NaCl-induced blood pressure rise. The combination of a
      tendency by the kidneys to retain NaCl together with a high NaCl intake
      can produce a blood pressure rise. This combination tends to cause NaCl
      retention, which can trigger blood pressure rises in susceptible humans
      and animals. Such blood pressure rises can augment renal NaCl excretion
      and regain the previous NaCl balance. In Dahl salt-sensitive rats several
      renal abnormalities encourage sodium retention. By analogy, renal
      "abnormalities" are probably present in people susceptible to
      hypertension.
AD  - Department of Internal Medicine, University of Minnesota Hospital,
      Minneapolis 55455, USA. tobia001@maroon.tc.umn.edu
FAU - Tobian, L
AU  - Tobian L
LA  - eng
GR  - HL 17871/HL/NHLBI
PT  - Journal Article
PL  - UNITED STATES
TA  - Am J Clin Nutr
JT  - The American journal of clinical nutrition.
JID - 0376027
RN  - 0 (Potassium, Dietary)
RN  - 0 (Sodium Chloride, Dietary)
SB  - AIM
SB  - IM
MH  - Animals
MH  - Cerebral Infarction/etiology/mortality
MH  - Cerebrovascular Disorders/etiology/mortality
MH  - Humans
MH  - Hypertension/*etiology/mortality/physiopathology
MH  - Kidney/physiopathology
MH  - Potassium, Dietary/administration & dosage/*pharmacology
MH  - Research Support, Non-U.S. Gov't
MH  - Research Support, U.S. Gov't, P.H.S.
MH  - Signal Transduction
MH  - Sodium Chloride, Dietary/administration & dosage/*adverse effects
RF  - 21
EDAT- 1997/02/01
MHDA- 1997/02/01 00:01
PST - ppublish
SO  - Am J Clin Nutr 1997 Feb;65(2 Suppl):606S-611S.

PMID- 8931843
OWN - NLM
STAT- MEDLINE
DA  - 19970218
DCOM- 19970218
LR  - 20051116
PUBM- Print
IS  - 0895-7061 (Print)
VI  - 9
IP  - 11
DP  - 1996 Nov
TI  - Introduction to the symposium: the sympathetic nervous system really is a
      key element in hypertension, with treatment implications.
PG  - 111S-112S
AD  - Department of Medicine, University of Minnesota, Minneapolis 55455, USA.
FAU - Tobian, L
AU  - Tobian L
FAU - Bunn, P
AU  - Bunn PA JR
LA  - eng
PT  - Journal Article
PL  - UNITED STATES
TA  - Am J Hypertens
JT  - American journal of hypertension :  journal of the American Society of
      Hypertension.
JID - 8803676
SB  - IM
MH  - Animals
MH  - Humans
MH  - Hypertension/*etiology/therapy
MH  - Sympathetic Nervous System/*physiology
RF  - 4
EDAT- 1996/11/01
MHDA- 1996/11/01 00:01
AID - 0895706196004256 [pii]
PST - ppublish
SO  - Am J Hypertens 1996 Nov;9(11):111S-112S.